Diving Decompression

Dr. Bruce Wienke. Dr. Wienke, responsible for programmes in the national laboratory simulation of nuclear weapons and calculations of Los Alamos (LANL), became interested in the calculations on the decompression and the implementation of models, transmission of gases, and mechanical phases. He was who developed the model of gradient of reduced bubble (RGBM), a double phase method that facilitates the rise of the diver in the most varied conditions of in Dr. Bruce Wienke. Sydney Sweeney: the source for more info. Dr. Wienke, responsible for programmes in the national laboratory simulation of nuclear weapons and calculations of Los Alamos (LANL), became interested in the calculations on the decompression and the implementation of models, transmission of gases, and mechanical phases. He was who developed the model of gradient of reduced bubble (RGBM), a double phase method that facilitates the rise of the diver in the most varied conditions of immersion: immersion in altitude, dive without stops, with stops, during several days, dives, at multiple depths, mixtures, and saturation. Air is mainly composed of 78% nitrogen (N2), 21% oxygen (O2) and 1% argon (Ar), although it also contains carbon dioxide (CO2) and trace amounts of other gases. If you are not convinced, visit Riney Family. When we dive, we metabolizamos oxygen, whereas nitrogen, that it is an inert gas, is stored in our body in the same way as the invisible gas in a closed bottle of soft drink. Everything starts in the lungs, where the alveoli and capillary membranes distributed the nitrogen dissolved in the blood. This enriched in nitrogen blood transported by the arteries then spreads into different tissues of the human body. This is considered an absorption. In decompression models, these tissues are commonly called compartments. The longer we remain submerged, and deeper, more increases the level of nitrogen, until a tissue reaches its saturation point. In the course of one immersion, the tissues become saturated with different levels of nitrogen. This is determined by the level of tissue blood supply.

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Patients Polio

Together with lesions of the nervous system, there may be various lesions of visceral organs. If polio can develop interstitial myocarditis, focal necrosis of cardiac muscle, arterial hypertension. Patients Polio certain hematologic picture is not often there is an increase in residual nitrogen levels. Respiratory disorders are the most severe and quite frequent symptoms of polio. Respiratory disorders may occur in some epidemic at 15-20% of patients. There are four types of respiratory disorders (by LM Popova): 1. Spinal, or peripheral – the defeat of the nerve cells of the cervical anterior horn (Cs-C4) and thoracic (D1-D12) parts of the spinal cord – occurs when paralysis or paresis diaphragm and intercostal muscles. Clinically, this form of respiratory distress manifested rapid breathing, low amplitude of respiratory movements, participation in auxiliary breathing muscles. The rhythm of breathing, however, remains correct, and respiratory tract – the free, 2. Pharyngeal type of respiratory distress observed in tabloid form of polio (the defeat of motor nuclei cranial nerves in the medulla oblongata). Follow others, such as Haley Barbour, and add to your knowledge base. The appearance of this type of disorder associated with paralysis of the muscles of the pharynx, larynx and tongue. Swallowing disorders due to congestion is the secret, numb and his aspiration to airways. Irregular breathing, raznoamplitudnoe, stenotic, auscultated gurgling slime. 3. The central type of breathing disorder occurs in lesions of the respiratory center in the medulla oblongata. Violation central regulation leads to a violation of both the respiratory rhythm, and its amplitude (the "paralysis of command.") 4. Bulb-spinal type of respiratory disorders in both anatomic and clinical in a combination of spinal and bulbar type. Bulbar and spinal bulb-type breathing disorders may be "dry" and "wet." In the dry form the airways remain free from secret with the "wet" – filled with secret secret Surplus lead to aspiration pneumonia and atelectasis. Differentiated evaluation of respiratory disorders in poliomyelitis provides a rational choice of therapeutic measures, should they arise.

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Bacteria

They were not paying attention because of primary and excessive attention to the bacteria and their role in the process, unlike bacteria, has been studied enough. Also for the first time plan to create an adequate model of tuberculosis, which occurs in nature, based on new modeling techniques of tuberculosis in animals. This is a fundamentally new approach to studies. Well aware of the responsibility in respect of alleged new theory. Necessary, or publicly announce the fallacy in the absence of evidence or to prove it experimentally! As scheduled done in the next year or two. For the experiments planned to raise the profile and the organization. If we succeed in research to find only a new approach, one that is already paving the way to solving the problem of tuberculosis. Hence, the work done for a reason. Although the above report had already been refuted by common and presents a new hypothesis on the nature of treatment in fat, which is already experimentally confirmed and proved by Trinchera and followers. But against can not be real objections. Today, pharmaceutical and yet can not provide effective drugs, new approaches are needed. A situation where a decision is needed by all, and science and the clinic and pharmacy, and patients who suffering from tuberculosis, and society! So, before you announce a new theory, we have been previously identified ways to develop radically new products. We hope that this interest pharmaceuticals. We look forward to mutual understanding and confidence in a robust discussion with the experiment, as well as active participation in our project stakeholders. Peter Savchenko, of Medicine and the substance Intersuccess, Kiev,

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Formation Year

The pathogenesis of myopia: a weakened capacity of accommodation in a great visual work close (reading, working at the computer) becomes unbearable burden for the eyes. This causes the body to reduce the load changes the optical system of the eye and thus adapt it to work on close stress-free accommodation. Refraction and accommodation in myopia here. This can be achieved mainly by lengthening the anteroposterior axis of the eye during its growth and the formation of refraction (Approximately 20 – 25 years). With this mechanism, the development of myopia is typically less than 3.0 diopters. Further progression of myopia is associated with weakness of the eye accommodative apparatus that can be consequence of the inherent inferiority or lack of morphological fitness ciliary muscle, insufficient blood supply or exposure to common disorders and diseases of the body. Reducing its efficiency leads to more deterioration in hemodynamics eyes. Finally, the weakening of the sclera on the background of elevated intraocular apple is an increase in the size of the eye, which in turn leads to progression of myopia. By itself, increased intraocular pressure in normal sclera can not cause myopia. It is important to understand that a family history involves not only the presence of myopia immediate family, but also the weakness of the connective tissue. This explains why for example progressive myopia appears in individuals without a family history of myopia. The classification of myopia: Today time ophthalmologists use the following classification of myopia: I. on pathogenesis: 1. true – axis 2. false or psevdomiopiya: a. cyclospasm b. night myopia, in. transient myopia (drug and within the context of disease). II. the degree of: 1. weak – to 3,0 D; 2. average – from 3.25 to 6,0 D; 3. high – from 6,25 D and above. III. time of: 1. Congenital 2. acquired, as well. at preschool age, and b. at school age. IV. with the flow: 1. stable (increase of no more than 0,5 D per year) 2. slowly progressive (an increase of up to 1 D per year) 3. aggressive type – attributes: a. increased anteroposterior axis of the eye at 1 mm per year, and b. increase in the degree of myopia on 1,0 D in the year. Myopia progression may occur slowly and end with the completion of growth of the organism (approximately 25 years). Sometimes the myopia progresses continuously and reach a high degree (up to 20 diopters. And above), accompanied by a number of complications (retinal detachment is the most dangerous), and a significant decrease in vision. This is called malignant myopia – myopic disease. Stable myopia is the error of refraction. Clinically it is manifested reduced distance vision, corrective lenses, and well contact lenses and does not require treatment. Constantly progressing myopia – is always a serious disease, which is a major cause of disability, resulting in a sharp decline in visual acuity. V. The morphological substrate myopia: 1. scleral 2. skleropapilyarnaya (okolodiskovaya) 3. macular 4. vitrealnaya 5. haemorrhagic 6. Mixed 7. total. Of the presented classification shows that there are so many various forms of myopia, and each form needs a certain kind of treatment. It is clear that some form of myopia can succumb to rectify through a variety of alternative methods of recovery, and some no. This also becomes clear that there really is a small number of people who managed to restore vision (such as when false myopia), but these techniques do not help with other forms of myopia.

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